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And this was to investigate the role of mid-kind in Alzheimer's. They found that the mid-kind in amyloid beta have similar patterns at the protein level. The researchers used a fluorescent sensor for amyloid beta assembly to show that the assemblies were broken up to the presence of mid-kind. Modeling of the data showed that mid-kind inhibits amyloid beta elongation. So we're copying it from accumulating. And secondary nucleation is where we have the beta amyloid. And it's starting to accumulate the material around it, like nucleation in a storm cloud. And so there's two specific phases during assembly formation. So they use nuclear magnetic resonance to confirm the finding. BEEP